Cadmium nitrate

236.42

237.87900

233-710-6

VF9RQV8VXV

3087|2570

C45896

White cubic crystals|White, amorphous pieces or hygroscopic needles|White, prismatic needles

126

0.56570

3.6 g/cm³

350 °C

132℃

In water, 156 g/100 g water at 25 deg C

Ambient temperature with open venting. /Tetrahydrate/

Oral-Rat LD50: 300  mg/kg; Oral-Mouse LD50: 100  mg/kg

Combustible with organic matter, reducing agent, combustible sulfur and phosphorus; heated to decompose toxic nitrogen oxide and cadmium-containing gas

Strongly hygroscopic|Enthalpy of Fusion: 18.3 kJ/mol at 360 °C|Specific gravity 2.455; bp 132 °C /Tetrahydrate/|... forms both a dihydrate and a tetrahydrate|For more Other Experimental Properties (Complete) data for CADMIUM NITRATE (9 total), please visit the HSDB record page.

Water soluble.

Nitrate and Nitrite Compounds, Inorganic

Strong Oxidizing Agent

Mixtures of metal/nonmetal nitrates with alkyl esters may explode, owing to the formation of alkyl nitrates; mixtures of a nitrate with phosphorus, tin (II) chloride, or other reducing agents may react explosively [Bretherick 1979 p. 108-109].

II

6.1(b)

UN 3082 9/PG 3

3

45-36/38-20/21/22-51/53-50/53-48/20/22-23-22-61-60-46-20/22

53-45-36-26-36/37-60-61

T,N,Xn

The warehouse is ventilated, low temperature and dry; lightly loaded and unloaded; stored separately from organic materials, reducing agents, sulfur, phosphorus and flammable materials, food raw materials

Mixed with reducing agent, sulfur, phosphorus, etc., heated, impacted, friction can be explosive

Stable during transport. /Tetrahydrate/

P201-P273-P308 + P313-P391-P501

H340-H350-H360FD-H373-H411

Generators of waste (equal to or greater than 100 kg/mo) containing this contaminant, EPA hazardous waste number D006, must conform with USEPA regulations in storage, transportation, treatment and disposal of waste.|Generators of waste (equal to or greater than 100 kg/mo) containing this contaminant, EPA hazardous waste number D006, must conform with USEPA regulations in storage, transportation, treatment and disposal of waste. /Cadmium/|SRP: At the time of review, criteria for land treatment or burial (sanitary landfill) disposal practices are subject to significant revision. Prior to implementing land disposal of waste residue (including waste sludge), consult with environmental regulatory agencies for guidance on acceptable disposal practices.

National Toxicology Program. Eleventh Report on Carcinogens (2005). The Report on Carcinogens is an informational scientific and public health document that identifies and discusses substances (including agents, mixtures, or exposure circumstances) that may pose a carcinogenic hazard to human health. Cadmium and Cadmium Compounds are listed as known to be human carcinogens. /Cadmium and Cadmium Compounds/[Available from, as of July 31, 2009: http://ntp.niehs.nih.gov/ntp/roc/eleventh/profiles/s028cadm.pdf]

Special Hazards of Combustion Products: Toxic oxides of nitrogen and cadmium oxide fume may form in fires. Behavior in Fire: Will increase intensity of fire when in contact with combustible material (USCG, 1999)

|Danger|H301 (85.11%): Toxic if swallowed [Danger Acute toxicity, oral]|P201, P202, P260, P261, P264, P270, P271, P273, P280, P281, P284, P301+P310, P301+P312, P302+P352, P304+P312, P304+P340, P308+P313, P310, P312, P314, P320, P321, P322, P330, P363, P391, P403+P233, P405, and P501|Aggregated GHS information provided by 47 companies from 6 notifications to the ECHA C&L Inventory. Each notification may be associated with multiple companies.|H301: Toxic if swallowed [Danger Acute toxicity, oral]|P201, P202, P260, P264, P270, P271, P273, P280, P281, P284, P301+P310, P302+P352, P304+P340, P308+P313, P310, P312, P314, P320, P321, P322, P330, P363, P391, P403+P233, P405, and P501|P201, P202, P260, P264, P270, P281, P301+P310, P307+P311, P308+P313, P314, P321, P330, P405, and P501|P201, P202, P264, P270, P281, P301+P310, P308+P313, P321, P330, P405, and P501

Excerpt from ERG Guide 141 [Oxidizers - Toxic]: As an immediate precautionary measure, isolate spill or leak area in all directions for at least 50 meters (150 feet) for liquids and at least 25 meters (75 feet) for solids. LARGE SPILL: Consider initial downwind evacuation for at least 100 meters (330 feet). FIRE: If tank, rail car or tank truck is involved in a fire, ISOLATE for 800 meters (1/2 mile) in all directions; also, consider initial evacuation for 800 meters (1/2 mile) in all directions. (ERG, 2016)

Excerpt from ERG Guide 141 [Oxidizers - Toxic]: Keep combustibles (wood, paper, oil, etc.) away from spilled material. Do not touch damaged containers or spilled material unless wearing appropriate protective clothing. Stop leak if you can do it without risk. SMALL DRY SPILL: With clean shovel, place material into clean, dry container and cover loosely; move containers from spill area. LARGE SPILL: Dike far ahead of spill for later disposal. (ERG, 2016)

Rubber gloves; safety goggles; dust mask (USCG, 1999)

If material on fire or involved in fire: Extinguish fire using agent suitable for type of surrounding fire. (Material itself does not burn or burns with difficulty.) Use water in flooding quantities as fog. Use "alcohol" foam, dry chemical or carbon dioxide. /Cadmium compounds, NOS/

Cadmium nitrate will increase the intensity of fire when in contact with combustible material. /Tetrahydrate/|Mixtures with wood or other combustibles may catch fire. /Tetrahydrate/

Stop discharge if possible, isolate and remove discharged material. /Tetrahydrate/

May be dangerous if it enters water intakes; notify local health and wildlife officials; notify operators of nearby water intakes. /Tetrahydrate/|In case of spill: notify local ... pollution control agencies. /Tetrahydrate/|SRP: Contaminated protective clothing should be segregated in such a manner so that there is no direct personal contact by personnel who handle, dispose, or clean the clothing. Quality assurance to ascertain the completeness of the cleaning procedures should be implemented before the decontaminated protective clothing is returned for reuse by the workers. All contaminated clothing should not be taken home at end of shift, but should remain at employee's place of work for cleaning.

/GUIDE 154: SUBSTANCES - TOXIC AND/OR CORROSIVE (NON-COMBUSTIBLE)/ First Aid: Move victim to fresh air. Call 911 or emergency medical service. Give artificial respiration if victim is not breathing. Do not use mouth-to-mouth method if victim ingested or inhaled the substance; give artificial respiration with the aid of a pocket mask equipped with a one-way valve or other proper respiratory medical device. Administer oxygen if breathing is difficult. Remove and isolate contaminated clothing and shoes. In case of contact with substance, immediately flush skin or eyes with running water for at least 20 minutes. For minor skin contact, avoid spreading material on unaffected skin. Keep victim warm and quiet. Effects of exposure (inhalation, ingestion or skin contact) to substance may be delayed. Ensure that medical personnel are aware of the material(s) involved and take precautions to protect themselves. /Cadmium compound/|/GUIDE 154: SUBSTANCES - TOXIC AND/OR CORROSIVE (NON-COMBUSTIBLE)/ Spill or Leak: ELIMINATE all ignition sources (no smoking, flares, sparks or flames in immediate area). Do not touch damaged containers or spilled material unless wearing appropriate protective clothing. Stop leak if you can do it without risk. Prevent entry into waterways, sewers, basements or confined areas. Absorb or cover with dry earth, sand or other non-combustible material and transfer to containers. DO NOT GET WATER INSIDE CONTAINERS. /Cadmium compound/|/GUIDE 154: SUBSTANCES - TOXIC AND/OR CORROSIVE (NON-COMBUSTIBLE)/ Fire: Small fires: Dry chemical, CO2 or water spray. Large fires: Dry chemical, CO2, alcohol-resistant foam or water spray. Move containers from fire area if you can do it without risk. Dike fire control water for later disposal; do not scatter the material. Fire involving tanks or car/trailer loads: Fight fire from maximum distance or use unmanned hose holders or monitor nozzles. Do not get water inside containers. Cool containers with flooding quantities of water until well after fire is out. Withdraw immediately in case of rising sound from venting safety devices or discoloration of tank. ALWAYS stay away from tanks engulfed in fire. /Cadmium compound/|/GUIDE 154: SUBSTANCES - TOXIC AND/OR CORROSIVE (NON-COMBUSTIBLE)/ Evacuation: ... Fire: If tank, rail car or tank truck is involved in a fire, ISOLATE for 800 meters (1/2 mile) in all directions; also, consider initial evacuation for 800 meters (1/2 mile) in all directions. /Cadmium compound/|For more DOT Emergency Guidelines (Complete) data for CADMIUM NITRATE (8 total), please visit the HSDB record page.

Listed as a hazardous air pollutant (HAP) generally known or suspected to cause serious health problems. The Clean Air Act, as amended in 1990, directs EPA to set standards requiring major sources to sharply reduce routine emissions of toxic pollutants. EPA is required to establish and phase in specific performance based standards for all air emission sources that emit one or more of the listed pollutants. Cadmium nitrate is included on this list.

The major hazards encountered in the use and handling of cadmium nitrate stem from its toxicologic properties. Toxic primarily by ingestion and inhalation, exposure to this odorless, colorless-to-white, crystalline substance may occur from its use in making other cadmium salts, in photographic emulsions, in coloring glass and procelain, and in the manufacture of nickel-cadmium batteries and turf fungicides. Effects from exposure may include headache, nausea, shortness of breath, chest pain, kidney damage, liver damage, emphysema, and intense pulmonary edema (possibly resulting in death). Processes and operations which may release cadmium fumes or dust should be enclosed and fitted with exhaust ventilation. In activities where overexposure is possible, workers should wear a high efficiency particulate filter respirator or self-contained breathing apparatus. Protective clothing also should be worn, including eye, face, and hand protection. All such clothing should be removed before leaving work. If contact should occur, immediately wash contaminated skin with large amounts of water. Do not eat, smoke, or drink in work areas. Cadmium nitrate presents only a moderate fire hazard (when in the form of dust) if exposed to heat, flame, or by chemical reaction with oxidizing agents, metals, hydrogen azide, zinc, selenium, or tellurium. Wear a self-contained breathing apparatus when fighting such fires. Cadmium nitrate should be stored in cool, well-ventilated areas, out of direct rays of sun, and away from combustibles and other fire hazards. Should hazardous concentrations of cadmium nitrate be released, first stop the discharge, then isolate and remove the discharged material. Before implementing land disposal of waste cadmium nitrate, consult environmental regulatory agencies for guidance.

D006; A waste containing cadmium may or may not be characterized as a hazardous waste following testing by the Toxicity Characteristic Leaching Procedure as prescribed by the Resource Conservation and Recovery Act (RCRA) regulations. /Cadmium/

D006; A solid waste containing cadmium may or may not become characterized as a hazardous waste when subjected to the Toxicity Characteristic Leaching Procedure listed in 40 CFR 261.24, and if so characterized, must be managed as a hazardous waste. /Cadmium/

highly toxic

In order to test the potential effect of prior exposure to different Cd concentrations on Cd uptake and accumulation, plants of Arabidopsis thaliana, including a phytochelatin-deficient mutant, cad1-3, and the wild type, were compared. For Cd uptake experiments, plants were grown for 1 week in nutrient solution containing different Cd concentrations (0, 0.05, 0.1, 0.25, 0.5, and 1.0 uM Cd(NO3)2). Thereafter they were subjected to 0.5 uM Cd labelled with (109)Cd for 2 hr. Uptake experiments with (109)Cd showed that the phytochelatin-deficient mutant cad1-3, accumulated less Cd than the wild type. Both a lower proportion and lower total amount of absorbed Cd were translocated to the shoot in cad1-3 plants compared to wild-type plants. Cadmium exposure also influenced the amounts of nutrients found, whereby after exposure to high Cd concentrations (0.5, 1.0 uM) during growth, cad1-3 roots contained less Fe, K, Mg, P, and S compared to roots of the wild type. In cad1-3 these elements decreased with increasing Cd concentration. The total Cd content in roots and shoots increased significantly with increasing Cd concentration during growth, although the increase was much less in cad1-3 plants. In time-dependent experiments of Cd uptake carried out between 15 and 120 min on plants not previously exposed to Cd, no significant difference in Cd accumulation between the mutant and wild type were found, although a smaller amount of Cd was translocated to the shoot in cad1-3 plants. The possibility that the differences in Cd accumulation in mutant and wild-type lines may be due to the cytosolic Cd regulation, which is inhibited by the complexation of Cd by phytochelatins, is discussed|The interaction between selenium and cadmium was studied in relation to cellular uptake and expressions of selenium-cadmium interaction. Human K-562 cells were pre-treated or simultaneously treated with (5 or 50 uM) selenite or (10 or 50 uM) selenomethionine and with (60 or 75 uM) cadmium nitrate. Cells pre or simultaneously treated with selenite revealed increased cadmium concentration with increased doses of selenite, particularly pronounced in the simultaneous treatments. In both treatments, selenium protection was observed during the exposure period, but not during the growth period. In cells simultaneously treated with selenomethionine and 60-uM cadmium, an increase in cadmium concentration was observed after increased selenium dose. In addition, it was found that simultaneous selenomethionine treatment with 60-uM cadmium resulted in selenium protection during the exposure period, although protection was not observed during the growth period.|Plants that hyperaccumulate metals are ideal subjects for studying the mechanisms of metal and mineral nutrient uptake in the plant kingdom. Indian Mustard (Brassica juncea) has been shown to accumulate moderate levels of Cd, Pb, Cr, Ni, Zn, and Cu. In this experiment, 10 levels of Cd concentration treatments were imposed by adding 10-190 mg Cd /per/ kg to the soils as cadmium nitrate [Cd(NO3)2]. The effect of Cd on phosphorus (P), potassium (K), calcium (Ca), magnesium (Mg), and the micronutrients iron (Fe), manganese (Mn), copper (Cu), and zinc (Zn) in B. juncea was studied. Plant growth was affected negatively by Cd, root biomass decreased significantly at 170 mg Cd /per/ kg dry weight soils treatment. Cadmium accumulation both in shoots and roots increased with increasing soil Cd treatments. The highest concentration of Cd was up to 300 mg /per/ kg dw in the roots and 160 mg /per/ kg dw in the shoots. The nutrients mainly affected by Cd were P, K, Ca, Fe, and Zn in the roots, and P, K, Ca, and Cu in the shoots. K and P concentrations in roots increased significantly when Cd was added at 170 mg /per/ kg, and this was almost the same level at which root growth was inhibited. Zn concentrations in roots decreased significantly when added Cd concentration was increased from 50 to 110 mg /per/ kg, then remained constant with Cd treatments from 110 to 190 mg /per/ kg. However, Zn concentrations in the shoots seemed less affected by Cd. It is possible that Zn uptake was affected by the Cd but not the translocation of Zn within the plant. Ca and Mg accumulation in roots and shoots showed similar trends. This result indicates that Ca and Mg uptake is a non-specific process.|We examined the influences of three trace metals on the accumulation of a major nutrient (NO3-) in Scenedesmus quadricauda. A comparative study on metal-nutrient interaction in free and immobilized states of algal cells was conducted. The effect due to interaction between different variables (cell state type, metal type, and metal dose) was studied to assess the variation in the nitrate uptake by free and immobilized cells. The results analyzed by ANOVA (three-way) (components: cell state type, metal type, and metal dose) confirmed that the inhibition of nitrate uptake by test metals was highly significant (P<0.001). Free and immobilized states of S. quadricauda responded differently (P<0.05, ANOVA) to the types of metal added. Uptake kinetics was studied by monitoring short-term uptake rates at different nutrient levels. Free and immobilized cells of the organism displayed noncompetitive modes of inhibition for Ni and Zn while a competitive mode of inhibition by Cd was observed in both free and immobilized states of the organism.|For more Interactions (Complete) data for CADMIUM NITRATE (6 total), please visit the HSDB record page.

LD50 Rat oral 300 mg/kg|LD50 Mouse oral 100 mg/kg

/AQUATIC SPECIES/ Daphnia magna (cladoceran) 0.17 ug/L/21 days in water hardness of 45 mg/L calcium carbonate, toxic effect: reproductive impairment.|/AQUATIC SPECIES/ Chilomonas paramecium (protozoan) exposed to 160 ug/L/48 hr, toxic effect: incipient inhibition.|/AQUATIC SPECIES/ Uronema parduezi (protozoan) exposed to 26 ug/L/20 hr, toxic effect: incipient inhibition.|/AQUATIC SPECIES/ Entosiphon sulcatum (protozoan) exposed to 11 ug/L/72 hr, toxic effect: incipient inhibition.|For more Ecotoxicity Excerpts (Complete) data for CADMIUM NITRATE (14 total), please visit the HSDB record page.

Salvinia natans (fern) exposed to cadmium nitrate for 21 days exhibited a bioconcentration factor of 960.|Mya arenaria, (soft shell clam) exposed to cadmium nitrate for 70 days exhibited a bioconcentration factor of 160.|Crassostrea virginica, (American oyster) exposed to cadmium nitrate for 98 days exhibited a bioconcentration factor of 1,220.|Lemna valdiviana (duck weed) exposed to cadmium nitrate for 21 days exhibited a bioconcentration factor of 603.

An adsorption rate of 1.22/hr in river sediments from the Nagara River, Japan, which is heavily impacted by industrial effluents and urban wastes, has been reported for cadmium nitrate; the desorption rate was reported as 0.0038/hr. These values correspond to a concentration factor of 321(1).

According to the 2006 TSCA Inventory Update Reporting data, the number of persons reasonably likely to be exposed in the industrial manufacturing, processing, and use of cadmium nitrate is 1 to 99; the data may be greatly underestimated(1).

A meta-analysis of existing scientific literature recently suggested that Cd is absorbed more efficiently by sheep if it is in the organic form in grass, than if it is added as an inorganic supplement to the diet. We tested this experimentally by feeding sheep grass from contaminated soil, compared with uncontaminated grass and with Cd added to the diet. To produce contaminated herbage, Cd nitrate was added to soil in 11 lysimeters sown with perennial ryegrass, with a further 11 lysimeters receiving no Cd to produce uncontaminated herbage. In the Cd-treated lysimeters, soil had increased exchangeable K, Mg, and Ca, leachate had increased K, Mg, Ca, Na, and P, grass had increased Cd and reduced Mg, Na, P, Mn, Fe, Cr, Al, and Ni, and there was some reduction in grass yield compared with untreated lysimeters. Grass from Cd-treated or untreated lysimeters was fed to groups of 12 ewes for 2 days, with Cd intake equated by adding Cd nitrate to the concentrate feed of ewes receiving the uncontaminated grass. The apparent absorption of Cd, Zn, Mo, Cr, and Al was increased for ewes receiving Cd-enriched grass, and apparent absorption of Cu was reduced, compared to those receiving supplementary inorganic Cd. Most of the unabsorbed Cd was excreted in feces within 4 days of feeding. The ewes consuming Cd in grass had increased B concentrations in their urine, possibly due to adverse effects of Cd on kidney function. Finally, the ewes were offered a choice of the two herbages and they ate significantly more of the uncontaminated grass. It is concluded that the apparent absorption of Cd and other heavy metals by sheep in a short-term experiment was greater when Cd was in the grass than when the Cd was added in in an inorganic form and that sheep partially avoided herbage with a high Cd concentration.

Cadmium nitrate has a purity of greater than 99%, with typical impurities including: chloride (0.005%), sulfate (0.005%), copper (0.005%), iron (0.002%), lead (0.005%), zinc (0.05%), and arsenic (0.001%).

Inhalation of fumes can produce coughing, chest constriction, headache, nausea, vomiting, pneumonitis. Chronic poisoning is characterized by emphysema and kidney injury. Ingestion causes gastrointestinal disturbance and severe toxic symptoms; both kidney and liver injuries may occur. Contact with eyes causes irritation. (USCG, 1999)|Carcinogens

INHALATION: remove patient to fresh air; seek medical attention. INGESTION: give large amounts of water and induce vomiting; give milk or egg whites; seek medical attention. EYES: flush with copious amounts of water for 15 min.; consult a physician. SKIN: wash with soap and water. (USCG, 1999)

Immediate first aid: Ensure that adequate decontamination has been carried out. If patient is not breathing, start artificial respiration, preferably with a demand-valve resuscitator, bag-valve-mask device, or pocket mask, as trained. Perform CPR as necessary. Immediately flush contaminated eyes with gently flowing water. Do not induce vomiting. If vomiting occurs, lean patient forward or place on left side (head-down position, if possible) to maintain an open airway and prevent aspiration. Keep patient quiet and maintain normal body temperature. Obtain medical attention. /Cadmium and Related Compounds/|Basic treatment: Establish a patent airway (oropharyngeal or nasopharyngeal airway, if needed). Suction if necessary. Watch for signs of respiratory insufficiency and assist ventilations if necessary. Administer oxygen by nonrebreather mask at 10 to 15 L/min. Monitor for pulmonary edema and treat if necessary ... . Anticipate seizures and treat if necessary ... . For eye contamination, flush eyes immediately with water. Irrigate each eye continuously with 0.9% saline (NS) during transport ... . Do not use emetics. For ingestion, rinse mouth and administer 5 ml/kg up to 200 ml of water for dilution if the patient can swallow, has a strong gag reflex, and does not drool. Administer activated charcoal ... . /Cadmium and Related Compounds/|Advanced treatment: Consider orotracheal or nasotracheal intubation for airway control in the patient who is unconscious, has severe pulmonary edema, or is in severe respiratory distress. Positive-pressure ventilation techniques with a bag valve mask device may be beneficial. Consider drug therapy for pulmonary edema ... . Monitor cardiac rhythm and treat arrhythmias if necessary ... . Start IV administration of D5W /SRP: "To keep open", minimal flow rate/. Use 0.9% saline (NS) or lactated Ringer's (LR) if signs of hypovolemia are present. For hypotension with signs of hypovolemia, administer fluid cautiously. Watch for signs of fluid overload ... .... . Treat seizures with diazepam or lorazepam ... . Use proparacaine hydrochloride to assist eye irrigation ... . /Cadmium and Related Compounds/

/ALTERNATIVE and IN VITRO TESTS/ Effects of some metals on the growth of cultured human erythroleukemia K-562 cells were investigated when grown in two different types of media based upon RPMI-1640 or Ham's F-10. The study on proliferation, using RPMI-1640 supplemented with sodium selenite, selenomethionine, mercuric chloride, methylmercuric chloride and cadmium nitrate showed no inhibition of growth at concentrations of 2.5, 25, 25, 2.5 and 25 uM, while at 75, 250, 50, 5 and 50 uM toxicity was apparent. Selenite at 5-50 uM and selenomethionine at 50-100 uM inhibited the growth. In Ham's F-10 supplemented with the same compounds no inhibition was found at concentrations of 5, 10, 25, 1 and 50 uM, while at 50, 100, 50, 5 and 75 uM toxic effects were noted. Selenite 10 uM and selenomethionine 25-50 uM inhibited the proliferation. Measurements of trace element levels in pellets of K-562 cells grown in RPMI-1640 or Ham's F-10 unveiled higher cell contents of cadmium and selenium in cells grown in RPMI-1640, being consistent with higher concentrations of these elements in that medium. Manganese and mercury concentrations were higher in cells grown in Ham's F-10 correlating with a higher medium concentration of these elements. The growth responses and cellular uptake differed between the metals and the selenocompounds and although extrapolating the results to humans is difficult the selenium exposures were in approximately the same order of magnitude as in human exposures. The compounds could be ranked according to decreasing toxicity as: methylmercuric chloride > mercuric chloride, cadmium nitrate, sodium selenite > selenomethionine.|/ALTERNATIVE and IN VITRO TESTS/ Recent reports, highlighting the relationships of cadmium exposure and vascular diseases, indicated that vascular endothelial cell was the target of cadmium (Cd) toxicity. However, the underlying mechanisms have not been fully elucidated. ...this study...evaluated the internalization of Cd(2+) into human umbilical vein endothelial cells (HUVECs) by a novel Cd(2+)-selective sensor suitable for living cells. Then, we detected apoptosis in the treated cells. Our results showed that Cd(2+) at low concentrations (< 10 umol/L) inhibited apoptosis induced by deprivation of serum and basic fibroblast growth factor (bFGF). To investigate the corresponding molecular mechanisms, we employed acridine orange staining and Western blotting of /microtubule-associated protein 1 light chain 3 alpha/ MAP1 LC3 to detect autophagy, and analyzed the levels of integrin beta4, caveolin-1 and activity of /phosphatidylcholine-specific phospholipase/ PC-PLC. Our results showed that low concentrations of Cd(2+) promoted autophagy and depressed the levels of integrin beta4, caveolin-1 and PC-PLC activity. The data suggested that autophagy played a key role in Cd(2+) induced endothelial dysfunction; integrin beta4, caveolin-1 and PC-PLC might be the targets of Cd(2+) in vascular endothelial cells

cadmium dinitrate

Grades: Technical; Reagent|Reagent grade: 99.0% assay /Cadmium nitrate, tetrahydrate/

Battery cathode|Nitric acid, cadmium salt (2:1): ACTIVE

Health Hazards -> Carcinogens